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Summary Literature (13)
Literature for DOID 0060224: atrial fibrillation


Xenbase Articles :
( Denotes literature images)
Cardiac expression and atrial fibrillation-associated remodeling of K₂p2.1 (TREK-1) K⁺ channels in a porcine model., Schmidt C,Wiedmann F,Tristram F,Anand P,Wenzel W,Lugenbiel P,Schweizer PA,Katus HA,Thomas D, Life Sci. March 3, 2014; 97(2):0024-3205.
Cloning, functional characterization, and remodeling of K2P3.1 (TASK-1) potassium channels in a porcine model of atrial fibrillation and heart failure., Schmidt C,Wiedmann F,Langer C,Tristram F,Anand P,Wenzel W,Lugenbiel P,Schweizer PA,Katus HA,Thomas D, Heart Rhythm. October 1, 2014; 11(10):1547-5271.
TASK-1 and TASK-3 may form heterodimers in human atrial cardiomyocytes., Rinné S,Kiper AK,Schlichthörl G,Dittmann S,Netter MF,Limberg SH,Silbernagel N,Zuzarte M,Moosdorf R,Wulf H,Schulze-Bahr E,Rolfes C,Decher N, J Mol Cell Cardiol. April 1, 2015; 81:1095-8584.
Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K2P) channels in murine disease models: focus on TASK-1., Wiedmann F,Schulte JS,Gomes B,Zafeiriou MP,Ratte A,Rathjens F,Fehrmann E,Scholz B,Voigt N,Müller FU,Thomas D,Katus HA,Schmidt C, Basic Res Cardiol. June 7, 2018; 113(4):1435-1803.
N-glycosylation-dependent regulation of hK2P17.1 currents., Wiedmann F,Schlund D,Voigt N,Ratte A,Kraft M,Katus HA,Schmidt C, Mol Biol Cell. June 1, 2019; 30(12):1939-4586.
Conservation and divergence of protein pathways in the vertebrate heart., Federspiel JD,Tandon P,Wilczewski CM,Wasson L,Herring LE,Venkatesh SS,Cristea IM,Conlon FL, PLoS Biol. September 1, 2019; 17(9):1545-7885.
Antiarrhythmic Properties of Ranolazine: Inhibition of Atrial Fibrillation Associated TASK-1 Potassium Channels., Ratte A,Wiedmann F,Kraft M,Katus HA,Schmidt C, Front Pharmacol. July 3, 2019; 10:1663-9812.
Electrophysiological effects of non-vitamin K antagonist oral anticoagulants on atrial repolarizing potassium channels., Wiedmann F,Schlund D,Kraft M,Nietfeld J,Katus HA,Schmidt C,Thomas D, Europace. September 1, 2020; 22(9):1532-2092.
Mechanosensitive TREK-1 two-pore-domain potassium (K2P) channels in the cardiovascular system., Wiedmann F,Rinné S,Donner B,Decher N,Katus HA,Schmidt C, Prog Biophys Mol Biol. January 1, 2021; 159:1873-1732.
Physical basis for distinct basal and mechanically gated activity of the human K+ channel TRAAK., Rietmeijer RA,Sorum B,Li B,Brohawn SG, Neuron. September 15, 2021; 109(18):0896-6273.
Inactivation influences the extent of inhibition of voltage-gated Ca+2 channels by Gem-implications for channelopathies., Allam S,Levenson-Palmer R,Chia Chang Z,Kaur S,Cernuda B,Raman A,Booth A,Dobbins S,Suppa G,Yang J,Buraei Z, Front Physiol. January 1, 2023; 14:1664-042X.
Electrophysiological Effects of the Sodium-Glucose Co-Transporter-2 (SGLT2) Inhibitor Dapagliflozin on Human Cardiac Potassium Channels., Müller ME,Petersenn F,Hackbarth J,Pfeiffer J,Gampp H,Frey N,Lugenbiel P,Thomas D,Rahm AK, Int J Mol Sci. May 23, 2024; 25(11):1422-0067.
KCNJ4 variants disrupt inward-rectifier potassium channel function and cause refractory epilepsy., Pan H,Liu D,Xu W,Li Y,Hu J,Henry O,Fuchs A,Jamra RA,Liu Z,He M,Chen Y,Wu S,Dong X,Chen Y,Wang P,Gu W,Jing H,Tang Y,Wang YJ,Mao X,Xiao N, Epilepsia. March 14, 2026; :1528-1167.